Thus, similar to spaceflight, the effects on human physiology as a result of prolonged exposure to a bed rest environment are multifactorial. A combination of sensorimotor alterations, reduced muscle strength, or presyncopal symptoms caused by orthostatic intolerance may inhibit timely execution of the egress.īed rest is a well-accepted spaceflight analog ( 8) to understand the implications of muscle disuse ( 9), cardiovascular deconditioning ( 10), and to simulate the axial unloading experienced by the sensorimotor system ( 11). For example, mobile mission tasks after landing on a planetary surface may include a rapid egress from a vehicle. These adaptations are often manifested in balance and gait disturbances ( 1, 2), cardiovascular deconditioning ( 3, 4), and loss of muscle mass, muscle coordination, and strength ( 5– 7). Multiple studies have demonstrated that exposure to spaceflight produces adaptations in sensorimotor, cardiovascular, and neuromuscular systems that are maladaptive upon return to 1 g. Physiological responses to microgravity have been studied over the history of human spaceflight.
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